Vale, Sancha Helena de LimaMarinho, Kívia Maria BatistaCunha, Thais AlvesGrilo, Evellyn CâmaraMiranda, Carolinne Thaisa de Oliveira FernandesBrandão-Neto, JoséVermeulen-Serpa, Karina Marques2025-06-182025-06-182021-12MARINHO, Kívia Maria Batista; CUNHA, Thais Alves; GRILO, Evellyn Câmara; MIRANDA, Carolinne Thaisa de Oliveira Fernandes; BRANDÃO NETO, José; SERPA, Karina Marques Vermeulen; VALE, Sancha Helena de Lima. Citocinas inflamatórias na distrofia muscular de Duchenne: uma revisão narrativa. Revista Eletrônica Acervo Saúde, [S.l.], v. 13, n. 12, p. 1-9, 21 dez. 2021. DOI: 10.25248/reas.e8992.2021. Disponível em: https://acervomais.com.br/index.php/saude/article/view/8992/5720. Acesso em: 15 jul. 2024.2178-2091https://repositorio.ufrn.br/handle/123456789/64007Objective: To describe the findings in the literature about inflammatory markers in Duchenne Muscular Dystrophy (DMD) and understand what is its association with disease progression. Bibliographic review: DMD is a severe genetic myopathy, characterized by an orthopedic, cardiac, and respiratory impairment, were it leads to the early death of the individual. Inflammation has a fundamental role in the pathophysiology of DMD. Elevated levels of cytokines and inflammatory markers, such as IL-1β, TNF-α, IL-6 and IL-17, activators and recruiters of inflammatory cells, seem to be related to the progression of the disease. Final considerations: Serum, tissue, and muscle levels of inflammatory markers were described as high in relation to healthy individuals. The association between inflammatory markers and the progression of DMD seems to be a peculiarity of the disease. Understanding this relationship is important for the establishment of new therapeutic approaches aimed at DMD. However, there is still a shortage on the subject in the literature. Therefore, there is a need for further related studiesen-USInterleucina-1Interleucina-6Interleucina-17Fator de necrose tumoral alfaMediadores da inflamaçãoInterleukin-1Interleukin-6Interleukin-17Tumor necrosis factor alphaInflammation mediatorsarticle